The "g-spot" is not a myth. Do you all know about this?
The "g-spot" is not a myth. Do you all know about this? ================Well ... its juices have been discussed in detail over on the "for the guys" board, so it MUST be true. :-)Diane- spotty on her g-spot spotting
The "g-spot" is not a myth. Do you all know about this? Have you tried Didi Seven?BK
The "g-spot" is not a myth. Do you all know about this?Well, that answers the "Where's VeeDub been?" question.LCK
The "g-spot" is not a myth.Do you all know about this? Yes. Though some of us find out later rather than sooner. But far far better later than never!s
Yes. Though some of us find out later rather than sooner. But far far better later than never!My whole world has changed. Thank you, evolution.
The "g-spot" is not a myth. Do you all know about this? Yes. (or should that be yes ... YES ...YES)-D
Do you all know about this? Yuh huh.
My whole world has changed. Thank you, evolution. AND, it's not just for fun anymore:"Is the G spot just for pleasure or does it have adaptive significance? An extensive series of studies in laboratory rats demonstrated that vaginal mechanical stimulation produced a strong pain blocking effect, stronger than 10 mg of morphine per kg of body weight. However, the most convincing evidence that vaginocervical stimulation blocks pain requires a verbal confirmation from women.Consequently, we performed a series of studies in women, measuring pain thresholds during vaginal self-stimulation.Pain thresholds were determined by applying a gradually increasing force to teach finger of one hand using a Ugo Basile Analgesia Meter. The subject places one finger on the 1 mm diameter point of the analgesia meter and a controlled, steadily increasing force is applied ranging from 0 grams to a maximum of 1 kg. The subject reports by saying "now," when the finger pain is first perceived (defined as "pain detection threshold") and by saying "stop," when finger pain becomes too uncomfortable to continue (defined as "pain tolerance threshold"). The pressure device is lifted from the finger when the subject says stop. Tactile thresholds are determined by applying a graded series of nylon monofilaments of varied stiffness (von Frey fibers) to the dorsal surface of the hand.We found that the elevation in pain detection threshold increased by a mean of 47% when pressure was self-applied to the anterior vaginal wall (the Grafenberg spot). When stimulation was applied in a pleasurable manner, the pain threshold was greater (by 84%) than that in the resting control condition. The PD threshold increased by a mean of 107% when the women reported orgasm.There were no increases in tactile (or touch) thresholds. This demonstrates that the effect was analgesic not an anesthetic effect and not a distracting effect. This analgesic effect was produced by pressure and by pleasurable self-stimulation applied to the anterior vaginal wall (G spot). (this was published in the Journal Pain., 1985, by Whipple and Komisaruk)Pleasurable self-stimulation, but not pressure applied to other genital regions also produced an analgesic effect. (Published in Journal of Sex Research., 1988, Whipple and Komisaruk) It was then demonstrated that an analgesic effect also occurs naturally during labor. We believe that childbirth would be more painful without this natural pain blocking effect, which is activated when the pelvic and hypogastric nerves are stimulated as the cervix dilates and from pressure in the vagina produced by the emerging fetus. (Published in International Journal of Nursing. Studies, 1990, Whipple et al)"http://www.hisandherhealth.com/articles/Beyond_the_G_spot_Where_do_we_go_from_here.shtml
This analgesic effect was produced by pressure and by pleasurable self-stimulation applied to the anterior vaginal wall (G spot). (this was published in the Journal Pain., 1985, by Whipple and Komisaruk)Interestingly, she found later on that eating a capsaicin-rich diet (capsaicin is what puts the hot in hot peppers) completely abolishes any analgesic effect. Not the capacity for vaginal pleasure, just any associated decrease in pain. (In the 1989 Physiology & Behavior.)About their conclusion in that 1990 article on analgesia during labor -- ...this natural pain blocking effect...is activated when the pelvic and hypogastric nerves are stimulated as the cervix dilates and from pressure in the vagina produced by the emerging fetus -- that "pressure in the vagina" sure as hell isn't G-spot joy!!sheila
Interestingly, she found later on that eating a capsaicin-rich diet (capsaicin is what puts the hot in hot peppers) completely abolishes any analgesic effect.I remember hearing about the studies of women who ate diets high in capsaicin experiencing more pain in labor and my heart fell. You'd think that anyone who could handle that much capsaicin would have a higher pain threshold. Crap!Ooh! I wonder: if someone who was given analgesic ate a sithload of spicy food, would the pain return? Does it "abolish any analgesic effect" in all cases or just a grapefruit squeezing through a pencil-sized hole?Hm...
Ohh! 2: "Capsaicin is being used in an analgesic agent in the treatment of painful disorders, causing long-term loss of responsiveness because it kills off the nociceptor, or it destroys the peripheral terminals.""Topical application of capsaicin relieves pain and itching by acting on sensory nerves. Capsaicin temporarily depletes “substance P”, a chemical in nerves that transmits pain sensations. Without substance P, pain signals can no longer be sent. . . . With the aid of a healthcare professional, capsaicin administered via the nose may also be a potentially useful therapy for cluster headaches."This one's pretty cool:http://www.nature.com/nsu/001026/001026-11.html
You'd think that anyone who could handle that much capsaicin would have a higher pain threshold.The whole issue with capsaicin gets more complex when you look at topical use--because then it's an analgesic! It acts by atrophying (temporarily) certain pain nerve endings, C-fiber nerve endings that secrete something called "substance P." The nerve endings atrophy, no more substance P, and no more pain. It's in some topical arthritis creams, for example. And that's why people who eat a lot of hot foods need 5-alarm chili before they'll call it spicy. But once you stop using it topically, those nerve endings are restored, and so are the sensations.The system of C-fibers--very thin fibers that lack the covering on most of our nerves--is extremely fascinating. They respond to emotional and tactile stimuli, and have psychological as well as sensory impact. Some of the scientists who study it feel it embodies the link between mind and body.But that's another story.sheila
Capsaicin temporarily depletes ?substance P?Hey....Good work, MissA!I hadn't mentioned that initial step, where basically the intense stimulus causes the C-fiber nerve endings to shoot their load, so to speak, and then they have nothing left. If you continue using the capsaicin cream regularly, the long-term effect is temporary atrophy of the nerve endings so no more substance P is produced. The language used in that piece you found--"killing the nociceptor" and "destroying the peripheral terminal"--exaggerates things and gives the erroneous impression that the effect is permanent.sheila
Thank you, ladies, for the additional information. Is the capsaicin connection the same reason why altoids and listerine strips work the way they do?
I don't understand. Are you all saying I should sit on Redsavina?6
Are you all saying I should sit on Redsavina?I don't know, I've been out of the loop. But anything's worth a try.
Is the capsaicin connection the same reason why altoids and listerine strips work the way they do? I don't think peppermint has capsaicin; it does have volatile oils of some kind, though, that's fer damn sure.See also: toothpaste, cinnamon candies, various flavored alcohols.
Is the capsaicin connection the same reason why altoids and listerine strips work the way they do?No no no! Capsaicin on very sensitive tissues would be a major turn-off. Called pain.sheila
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